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The nurse understands that the compensatory physiologic mechanism that leads to altered urinary output is release of salt and water retention-causing hormones antidiuretic hormone (ADH) and aldosterone.

The reabsorption of sodium and water by the renal tubules is accelerated by the release of aldosterone and ADH, which results in a reduction in urine production. Renal artery vasoconstriction results from SNS stimulation. Although -receptor stimulation raises cardiac output, this would also increase urine output. Fluid seeps into the interstitial area during shock from the intravascular space.

What is the primary factor that leads to hypovolemic shock?

Blood loss from significant injuries or major blood vessel ruptures is the most frequent cause of hypovolemic shock. Hemorrhagic shock is what this is. Additionally, it can be acquired by burns, acute vomiting, or even bleeding heavily during pregnancy.

What do examples of hypovolemic shock mean?

Significant fluid (other than blood) loss can lead to hypovolemic shock. Refractory gastroenteritis and severe burns are two conditions that can cause hypovolemic shock as a result of fluid loss.

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A patient with hypovolemic shock has a urinary output of 15 ml/hr. The nurse understands that the compensatory physiologic mechanism that leads to altered urinary output is

a. activation of the sympathetic nervous system (SNS), causing vasodilation of the renal arteries.

b. stimulation of cardiac β-adrenergic receptors, leading to increased cardiac output.

c. release of aldosterone and antidiuretic hormone (ADH), which cause sodium and water retention.

d. movement of interstitial fluid to the intravascular space, increasing renal blood flow.

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