Although it is clear that a blood clot in the coronary artery network can result in a heart attack or coronary, it is debatable whether or not the coronary arteries are considered to be a component of the heart. Additionally, blood clots may develop inside the heart in persons who have specific cardiac rhythm issues, such as general arrhythmia or atrial fibrillation. The greatest danger in this situation is that bits of a blood clot might separate and go to the lungs, where they could result in a pulmonary embolism.
How does blood clot?
Blood is actually quite a fascinating stuff, it turns out. As most of us are aware, it supplies nutrients to cells and removes waste produced as a result. Additionally, a very sophisticated immune response to hordes of invaders that would kill us without that reaction is mediated by white blood cells and a variety of proteins. In terms of coagulation, it starts when platelets come into contact with a damaged vessel wall lining. This causes a series of protein changes that lead to the development of fibrin, the gelatinous material that we know as a clot. The intriguing point is that when the vascular system is interrupted, blood MUST produce a semi-solid clot and MUST generally stay a fluid. We would bleed out with even slight injuries or have unpredictable disruptions in the circulation, both of which are essential for survival. One of the great marvels of human physiology is the fact that this occurs the majority of the time. However, some protein abnormalities and clotting diseases, like hemophilia, can produce aberrations that upset this delicate equilibrium. Although some of the information here may be incorrect because I am not a biochemist, the academic vascular surgeon who educated me had this as his principal area of study focus.
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