The pathogenesis of A.F can be regarded as the interaction that exist between firing ectopic foci and atrial tissue substrate.
Pathogenesis of A.Fs serves as one in which there is initiating triggers, which do occur in a rapidly firing ectopic foci that is found in pulmonary veins and this triggers interact with the abnormal atrial tissue substrate that can maintain arrhythmia.
Typical atrial flutter can be referred to as counterclockwise movement that is found in reentrant circuit around the tricuspid valve annulus.
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