If vasopressin (adh) increases water reabsorption by the cortical and medullary collecting ducts of the nephrons, would vasopressin secretion be increased or decreased with dehydration?

Vasopressin (ADH) stimulates water reabsorption via the cortical and medullary gathering tubes of the nephrons, therefore it would rise with dehydration.

Antidiuretic hormone (ADH):

It is also known as vasopressin, which is a nonapeptide produced by specific neurons in the suprachiasmatic and paraventricular nuclei. These nuclei carry vasopressin to the posterior pituitary, where it is produced in response to rising plasma osmolality and falling blood pressure.
The hypothalamus contains osmoreceptors, and atrial stretched endings and artery baroreceptors also send signals to this area. Vasopressin's effects are mediated via the V1a, V1b, and V2 receptor subtypes. Vascular smooth muscle has V1a receptors that are connected to the phosphoinositide pathway.

ADH in the bloodstream has an impact on the cardiovascular and renal systems:

ADH increases the kidneys' reabsorption of water, which reduces the amount of urine formation.
Vascular smooth muscle contraction, arteriole constriction, and peripheral vasoconstriction are all brought on by Vasopressin. Palor is the result, and the coronary and brain arteries dilate as a result.

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