The impact of Nimodipine on cardiac contractile cells is that Ca[tex]2^{+}[/tex] would enter into the cell independent of electrical stimulation and would stimulate Ca[tex]2^{+}[/tex] release to produce more frequent cardiac muscle contractions.
The voltage-gated L-type calcium channels located on the smooth muscle cells of arterial blood arteries are what dihydropyridines bind to and block to function. Voltage-gated L-type calcium channels are ones that typically open in response to an electrical signal, often known as an action potential.
A calcium channel blocker, nimodipine. Calcium ions, which enter smooth muscle cells as slow ionic transmembrane currents after depolarization, are necessary for the contractile actions of these cells. It's likely that Nimodipine will improve the cardiovascular effects of other calcium channel blockers.
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