You are examining the DNA sequences that code for the enzyme phosphofructokinase in skinks and Komodo dragons. You notice that the coding sequence that actually directs the sequence of amino acids in the enzyme is very similar in the two organisms but that the surrounding sequences vary quite a bit. What is the most likely explanation for this? (a) Coding sequences arc repaired more efficiently. (b) Coding sequences are replicated more accurately. (c) Coding sequences arc packaged more tightly in the chromosomes to protect them from DNA damage. (d) Mutations in coding sequences are more likely to be deleterious to the organism than mutations in noncoding sequences.

Mutation are changes in the hereditary grouping, and they are a fundamental driver of decent variety among living beings. These progressions happen at a wide range of levels, and they can have broadly varying results. In natural frameworks that are equipped for proliferation, we should initially concentrate on whether they are heritable; explicitly, a few transformations influence just the person that conveys them, while others influence the entirety of the bearer creature's posterity, and further relatives
A single mutation can have a large Effect, yet by and large, developmental change depends on the amassing of numerous transformations with little impacts. Mutational impacts can be valuable, hurtful, or unbiased, contingent upon their unique circumstance or area. Most non-nonpartisan transformations are injurious
Hence, the right answer is option d "Mutations in coding sequences are more likely to be deleterious to the organism than mutation in noncoding sequences"