Question

How would the feedback mechanism function to restore homeostasis after high TSH levels? Include receptor details

Answer 1

Answer:

Immediate effect of high TSH is release of thyroxin.

Low TSH leads to release of low thyroxin.

Hypothalamus releases TRH (thyrotropin releasing hormone) and TRH acts on pituitary to release TSH.

Pituitary gland releases TSH.

TSH stimulates thyroid gland to release thyroxin.

Thus if there is low TSH in the blood, immediately, the pituitary enters into action releasing high TSH, TSH enters into action by triggering thyroid to release more thyroxin and thus maintains homeostasis of thyroxin within blood stream.

If there is high TSH in blood stream, immediately, the thyroid released more thryroxin, thus more thyroxin could be found in blood stream, high levels of TH (thyroxin) in blood signals pituitary to release less TRH and thus it triggers less TSH from hypothalamus, and thus less thyroxin again from thyroid gland. Thus high TSH in blood through low TSH and TH triggers to maintain homeostasis in blood.

Thus TH (thyroxin) has negative feedback on hypothalamus and pituitary glands.

Explanation:

Answer 2

The feedback mechanism is negative which is responsible for restoring homeostasis after high TSH levels.

The thyroid hormone is regulated by thyrotropin-releasing hormone gene which is a negative feedback. The hypothalamus is responsible for the release of thyrotropin-releasing hormone (TRH) that regulates the thyroid hormone. Thyroid hormone production is controlled by the hypothalamic-pituitary-thyroid axis.

Thyrotropin-releasing hormone (TRH) signals the anterior pituitary in order to release thyroid stimulating hormone which regulates the release of thyroid hormone so we can conclude that the feedback mechanism is negative which is responsible for restoring homeostasis after high TSH levels.

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